Wednesday, February 10, 2010

Wednesday, Feb 10, 2010 Face Lacerations

Vet Tip of the Day: Facial Lacerations
Key Words/Phrases: Wound healing, primary closure, golden period


We've been hammering limb anatomy and lameness for over a week, so I think it's time to take a break and look at something entirely different. Facial lacerations are a relaxing break out topic because while they often start with a horrified horse owner and a gaping wound such as this one, they are simple to treat and usually end with an excellent outcome.

The tissues of the face have a tremendous blood supply, and because blood is what carries all the magical factors necessary for wound healing to the site of injury, lacerations on the face have a healing advantage not present in other areas of the body.

The first six hours after a skin laceration occurs is called the "golden period" in wound healing.   In general, sutures placed in a wound more than 6 hours after its occurrence are likely to fail, no matter how carefully the wound is prepared and how correctly the sutures are placed.  With every minute that passes after a laceration is sustained, exposure to external factors increases the risk of infection while at the same time the lack of blood supply to exposed tissues decreases the chance that tissue will remain alive and reattach when sutured.  The one anatomical exception to the "golden period" rule applies to facial lacerations, where wounds often heal uneventfully when sutured 24 hours or more after they occur.  Even large wounds filled with debris that involve skin flaps with wide areas of exposed underlying bone usually heal well, due to the excellent blood supply to tissues of the face.

The horse pictured here sustained a high impact injury involving a rigid metal object. Following sedation with 10 mg of intravenous detomidine, and subcutaneous placement of a local anesthetic around the wound margins, this horse's wound was carefully cleaned with dilute betadine, then flushed copiously with sterile saline.   Skin staples were used to hold the skin flap back in position.  The staples were removed 14 days later and as you can see, the wound healed without any skin loss.  Even if you find a severe face laceration that is over 12 hours old (the thunderstorm during the night is a classic for this), don't despair - primary closure may still be possible and successful.

Horses that sustain lacerations to the face and have concurrent nose bleeds should always receive antibiotics.  The presence of bleeding at the nose indicates that the concussive force may have been severe enough to cause bleeding into one of the sinuses that underlie much of the space beneath your horse's forehead and nose.  Because the sinus cavities communicate with the nasal passages, bacteria in the nasal passages can gain access to the sinus.  If there are broken blood vessels in the sinus, bacterial infection (sinusitis) can set up housekeeping in the sinus cavity.  Treating a well established bacterial sinusitis is difficult, and the need to do so can be avoided by initiating antibiotic treatment at the time of facial trauma.  So don't forget - face lacerations accompanied by nose bleeds should be seen by your veterinarian, even if the laceration itself is not severe.

I'm late writing the Veterinary Tale for this week, so it may not get posted tomorrow, but I'll do my best to get it to you soon.  The practice website is being updated this week.  I really like the new look and will let you know as soon as it is on line so you can check it out and give us your opinion.

The sun was out today in Reno - halleluiah! We used to take it for granted, but after this winter, no longer.

Enjoy,
Chrysann

Tuesday, February 9, 2010

February 9, 2010 - Injectable Joint Therapies

Vet Tip of the Day: Injectable Joint Therapies
Key Words: Adequan, Legend, PSGAG, Hyaluronic Acid

To finish our segment on the hock, here is a brief discussion of the two FDA licensed injectable agents to improve joint health.  The first, Adequan, is used more commonly for osteoarthritis of the hock (bone spavin).  Adequan is a large molecule with the chemical name polysulfated glycosaminoglycan (PSGAG) which cannot be absorbed when given orally and so is administered by intramuscular injection.  Adequan has been shown repeatedly in controlled clinical trials to improve lameness associated with chronic degenerative joint disease, however, on an individual animal basis, it's effectiveness is quite variable.   Its mechanism of action is similar to chondroitin and glucosamine.  Remember that these oral supplements are building blocks used by the body in the construction of cartilage.  Adequan is a very large molecule, more complex than either chondroitin or glucosamine, and is further along the construction pathway in the formation of cartilage matrix.  The PSGAG molecule is incorporated into cartilage, giving it elasticity and shock abosorbing capacity.

I use the brand name Adequan, although there are generic PSGAG products available, as well as compounded products.  However, as with oral supplements, the molecular construction of the generics and compounded products are not necessarily equivalent to Adequan, and have not stood the test of clinical or experimental study.  In fact, Luitpold Pharmaceuticals, the manufacturer of Adequan, has spent quite a bit of money conducting quality research on their product, and in the comparison studies performed to date, Adequan has outperformed generics.  Once again, you get what you pay for.

Legend is injectable hyaluronic acid (HA).  And yes, just like PSGAG,  hyaluronic acid comes in many molecular weights and qualities, and so far, for intravenous administration, Legend is the gold standard.  While Adequan is used most commonly for hock arthritis, it is not uncommon to combine Adequan and Legend as joint therapies.  In the joint environment, hyaluronic acid and PSGAG have complex actions.  In addition to acting as structural components of joint fluid and cartilage, both HA and PSGAG have anti-inflammatory effects within the joint.  On top of that, when used together, each component enhances the activity of the other, so 1+1=3!  Therefore, in performance horses with joint issues, it may be advisable to use combination therapy. 

There is a standard protocol for initiation of Adquan therapy, established by the manufacturer.  The recommendation is to give 1 vial every 4 days for 28 days followed by one vial every 2-4 weeks thereafter. Legend is often given once monthly intravenously as maintenance therapy, with additional doses given around the time of competition.

Oral and injectable chondroprotectant agents are widely used in treating osteoarthritis of the hocks.  They often are used in conjunction with intra-articular (directly into the joint space) injections of similar products in combination with corticosteroids, as well as oral adminstration of non-steroidal anti-inflammatories such as phenylbutazone.  Finally, a topical anti-inflammatory cream is now available which provides local, temporary pain relief and can be effective to control hock pain during exercise.  This product is Surpass, or diclofenac acid.

We've covered a lot of information on the hock.  If you are interested in learning more,  use the key words provided as Search words in Google and you will find LOTS more to read.

Have a great day,
Chrysann

Monday, February 8, 2010

Monday, Feb 8 2010 - Winding up the Hock

Vet Tip of the Day: Oral Joint Supplements
Key words: chondroprotectant, synovial fluid, cartilage, joint capsule

Osteoarthritis is only one of many conditions that affect the hock; in the future we will come back and discuss other disease processes that affect tarsal joints.  But to conclude this introduction to hock anatomy and bone spavin, the most common cause of hock lameness, there will be two final Vet Tips of the Day: today we will look at oral joint supplements, how they work, and IF they work.  Tomorrow we will consider injectables.

It is important to distinguish between oral and injectable agents administered as chondroprotectants.  "Chondro" refers to cartilage, "protectant" is self-explanatory.  Chondroprotectant joint supplements are substances which claim to enhance the health of joints by improving the the quality of joint fluid and joint cartilage.  Cartilage is the spongey substance which lines the surface of bones within joints.  The cartilage is bathed in synovial fluid, or joint fluid, which is contained within the joint space by the joint capsule.  The joint capsule is lined by synovium, the site of joint fluid production.  In the simplest terms, the joint fluid and cartilage act as shock absorbers to diminish the force transmitted to bones when joints are compressed by weight bearing.

You've almost certainly heard of glucosamine and chondroitin.  These are two of the most common ingredients in oral chondroprotective joint supplements.  These substances are two of the primary building blocks the body uses in the construction of cartilage.  Theoretically, by increasing the amount and quality of glucosamine and chondroitin in the diet, the body is better able to produce and maintain healthy cartilage.  There are three major problems with this theory.  First, there are many structural varieties of glucosamine and chondroitin, and their absorption across the gut wall is widely variable.  Second, and perhaps more importantly, there are few government regulations regarding the manufacture of oral joint supplements, which are considered "neutraceuticals".  Therefore, the amount and quality of ingredients listed on the label vary widely from product to product, and there is not even any guarantee that the label claims of a given product are true.  Therefore, when buying an oral joint supplement, you are completely at the mercy of the manufacturer's ethics regarding the actual contents of the product.

Finally, the third problem is, assuming the product you buy is excellent quality and your horse absorbs the ingredients, there is very limited research availabe indicating what an appropriate dosage is for a given combination of chondroprotective ingredients.  In addition to glucosamine and chondroitin, additional additives found in many joint supplements include cetyl myristoleate (an anti-inflammatory) , hyaluronic acid (a major components of joint fluid), as well as various herbal extracts such as devil's claw, boswellia and avocado extract, just to name a few.

Before you throw in the towel, be comforted in knowing that, while limited, there are a few well designed clinical studies indicating that the oral administration of chondroprotective agents can improve lameness in horses with chronic osteoarthritis.  The bottom line is, at this time there is more unknown than known about these products, so if you choose to give one to your horse, buy a recognized brand manufactured by a company with a longstanding reputation.  Be sure the product is clearly labelled with respect to ingredients, amounts of ingredients, and recommended dosage.  It should be easy to calculate the actual amount of each ingredient you will be giving your horse by reading the label.  The product should have a lot number and expiration date easily identifiable, and there should be clearly printed information telling you how to contact the manufacturer.  Finally, if the product claims to cure everything from laminitis to navicular disease to hock pain in 30 days, don't buy it.  Remember, you get what you pay for, and if it sounds too good to be true, it probably is.

Sunday, February 7, 2010

Fri & Sat Feb 4,5. More on the hock

Vet tip of Two Days!
Key words: osteoarthritis, hock,chondoprotective, joint therapy, glucosamine, chondroitin

My apologies for this late vet tip. It's actually Sunday morning, but it is snowing again and I've get an hour before I go out to do a couple of calls this morning. One of them is to inject the carpus of an older rope horse with chronic osteoarthritis. Do you remember how many joints there are in the carpus and which joint on your body is similar anatomically to this equine joint? (for review see last Wednesday's blog).

Osteoarthritis of the lower hock joints (bone spavin) is the most common cause of chronic lameness that I see in my practice, with forelimb heel pain a close second. Bone spavin occurs in every type of athletic horse, and unfortunately it is not uncommon to see horses develop sore hocks at a relatively early age. The 3 or 4 year old cutting horse is the extreme example, but jumpers, dressage horses, foxhunters, endurance horses, and other western sport horses may have degenerative changes in their hocks before 10 years of age.

Why are these joints predisposed to arthritis more than other joints of the body? Probably because of their unique position of high stress and low motion. Watch your horse's hocks closely as he trots on the longe line. Most of the flexion that occurs is due to motion of the large tibiotarsal joint, which has a large joint space and round, ball bearing like sliding surface where the tibia sits on the knuckle shaped talus. This joint is an uncommon site for osteoarthritis secondary to athletic activity.

Now look at the radiograph from Thursday's blog (the normal hock), then look at this radiograph which demonstrates osteoarthritis in the distal intertarsal and tarsometarsal joints. These are the joints at the bottom of the hock that are complex and relatively rigid in construction. The joint space is the thin black line between the rows of tarsal bones. Imagine the force that flows through these joints when your jumper throws his thousand plus pound body into the air to clear an obstacle or your barrel horse digs in to accelerate out of the turn around the last barrel or your dressage horse sucks it up to sit back on his hocks and put that tremendous effort into a canter pirouette. There isn't much room for error in these joints, because there is very little room for movement, so over time, the stress of high force through tightly packed bone results in chronic inflammation.

Joint fluid lubricates and cushions the cartilage that lines bone surfaces. The inflammatory process causes this fluid to becomes thin and less able to absorb repeated impact. The cartilage, in turn, looses its flexibility and ultimately the bone is left to take up too much of the load of activity. It's response is to say "Hey, I'm working too hard, maybe if there is more of me this job will be easier." Unfortunately, the production of extra bone simply destroys the smooth architecture of the carefully constructed joint anatomy with the end result: pain.

That's how the process starts, now how do we treat it?

Many of the key words for this vet tip are related to the huge topic of joint therapies. Tomorrow's vet tip will be devoted to the subject of oral and intra-articular therapies, and trying to separate fact from fiction. To close today's entry, here is the bottom line: there is sound, repeatable scientific evidence that certain oral and injectable chondroprotective agents can help to alleviate the inflammation, and therefore the pain, associated with osteoarthritis of the hock. The efficacy of these products is variable, and the products themselves are variable. We will look at them in more detail tomorrow.

I hope you are enjoying these vet tips - we are in the process of updating the practice website - the blog name is probably going to change (high brow internet search engine stuff that is GREEK TO ME). I promise to keep everyone up to date as the High Desert Veterinarian continues to explore cyberspace in order to share the knowledge and wonderful life of a practicing veterinarian with you all.

Thursday, February 4, 2010

Thursday, Feb 4 - The Hock and a Veterinary Tale

Vet Tip of the Day:  The Hock
Key Words: Equine tarsus, horse hock anatomy, bone spavine, degenerative joint disease

Today we will look at the hock, which is the hind limb joint that sits above the cannon/splint bones.  It corresponds anatomically to your heel.  There are 4 joints in the hock (identify them on the radiograph).  From top to bottom they are the tibotarsal joint, the proximal and distal intertarsal joints, and the tarsometatarsal joints.

The two distal intertarsal and tarsometatarsal joints are low motion joints that are very common sites for arthritic changes (degenerative joint disease or bone spavin) in all athletic horses.  This is a very common, and very treatable condition. Most horses with bone spavin continue in full work with maintenance veterinary care in the form of intra-articular injections with corticosteroids and hyaluronic acid in conjunction with a regimen of oral or injectable chondoprotectant agents such as Adequan, Legend, or oral chondroitin sulfate/glucosamine products.

The hock is a complex joint and is the site for many different problems that can cause lameness.  We will spend a few days looking at it in more depth, but since today is a Veterinary Tale day, this is the end of this Vet Tip - here's the story - hope you enjoy it.

Chapter 2 Good News Bad News

Thursday was a good news/bad news day. In the truck, my cell phone in its normal position, wedged between my head and left shoulder, in what I consider my personal “hands free” mode, I’m speeding of course, steering with my left hand while rubbing my dog Sticky behind her ears with my right hand. Sticky is in her favorite spot, curled up beside me with her shoulder pushing into my right hip and her head in my lap. This pretty much describes how I spend the majority of my life – motoring from one appointment to the next, multi-tasking on the phone, and rubbing my dog’s ears because I don’t have time to meditate. One of my two veterinary hopeful college student assistants completes our travelling circus, doing her best to keep me on track as I zoom around trying to protect the well-being of the equine population of northern Washoe County. On this particular day I was on the phone with a third year medicine resident from UC Davis who was telling me that Brandy was on her way home, fully recovered after her experience dancing with a T-post. Good news.

Sticky has been my constant companion for the past 8 years – she is much cuter than me, with better manners and a distinctly more endearing personality. She’d been in the truck when I treated Brandy, but she knew better than to get into the middle of that mess, and slept through the drama on the front seat. My clients, without exception, adore Sticky, and there is an ongoing debate among them whether or not she is part coyote. The physical resemblance is remarkable, but her disposition is so quiet, consistent and loving that it is hard to imagine she is related to those conniving rascals that lure puppies out into the desert and rip them to shreds. I tell everyone Sticky is a PCD, or Prostitute Companion Dog, descended from the gentle dogs that were friends to the girls in Julia Bouillet’s Bordello in Virginia City at the turn of the century. Some folks find this confusing, but it works for me.

I met sticky 8 years ago while responding to a call from a new client. Tanya Buckman was a cowgirl with a lame foal. Tanya was about 40 years old, 5’7”, with hair so blond, teeth so white, breasts so perky and jeans so tight if I wasn’t in Nevada I wouldn’t have believed it possible. When I first moved west these women were a real challenge to me, and I still look at them with some puzzlement and more than a little envy. They all are so beautiful, and so NICE, and SO TOUGH, they belong in movies. Every one of them wears their womanhood like a ship’s flag: the big belt buckles, the white cowgirl hats, the long stride and the high heeled Ropers. How they get their Levi’s to bunch up just so over the tops of their boots is beyond me. I’m a Yankee, I grew up in New England, I wear a string of pearls every day and my idea of risque is leaving the top button on my polo shirt open.

I’m going to digress for a minute here, but just to give you an example of what these gals are like, here’s what happened when I volunteered to draw blood for pseudorabies testing from a 4-H project pig belonging to one of my cowgirl client’s daughters. When I arrived the pig was loose in a large pen and since my generosity stopped short of chasing it around like a lunatic I asked the daughter to catch Charlotte for me. I watched her futile attempts to contain the pig until my head was about to explode then announced my impending departure if the pig wasn’t under control within 5 minutes. The daughter pulled out her cell phone and made a quick call.

The next thing I know, a bright red 1 ton Dodge diesel pickup comes screaming down the driveway from the house to the barn where we waited with the pig. The truck stops in a cloud of dust, the driver’s door flies open and Raquel Welsh in skin tight Levi’s gets out, struts over the to the pig enclosure, climbs the fence, grabs the pig by a back leg so fast I never saw her move, and hollers over her shoulder at me, “Hurry up and bleed this damn thing, I’ve got to get my nails done at three o’clock.” That was my client, the cowgirl mom, and I guarantee you that when she sashayed away from that pig pen she smelled just as fresh as a daisy.

Tanya Buckman was one of these Nevadan women, and like so many of them she was a savvy hand with horses. She had a buckskin quarter horse mare which had foaled 2 weeks previously. Everything was fine, until yesterday when the filly’s leg became swollen and it began to have trouble walking. Tanya easily cornered the wary 100 pound youngster and held her while I began my examination, questioning her as I did.

“ Has the mare ever foaled before, Tanya?” I asked as I took the foal’s temperature.

“No, this is her first, and she was real nervous about letting it suckle for the first couple of days,” she replied. “Does she have a fever?”

“Her temp’s 102.5, which is a little higher than I’d like to see. Maybe she didn’t get enough colostrum when she was born.”

Colostrum is the special milk that mares only produce for a few days after the birth of a foal. Horses are born with what is called a “naïve” immune system. They have no antibodies to protect them against infection, and must drink enough antibody rich colostrums within the first 24 hours of life or they are at high risk of developing serious infections. One of the most common ways for such infection to manifest is as a septic joint, which causes severe lameness and can be life threatening if the infection invades the bone or spreads to other organ systems.

After palpating the fluid in the foal’s fetlock joint and asking a few more questions, I was certain that the most likely cause of the lameness and fever was a bacterial infection in the joint that needed immediate attention. Tanya was clear that she wanted the foal treated there on the farm, so we began preparations to flush the joint. Needles are inserted in 2 locations, one to allow delivery of sterile fluid that lavages the joint space, the second to provide an exit route for the infected joint fluid and bacteria. This procedure is ideally performed under sterile, or aseptic, conditions. This presents a challenge in a field setting. The trick is not to make matters worse by introducing more bacteria into the joint than already are present.

The best environment Tanya could offer was a 3 sided shed with manure rich shavings covering a dirt floor. On our side was the fact that there was very little wind. In general I love the climate in northern Nevada – we enjoy over 300 sunny days a year and while winter is winter, the sun and 5,000 foot elevation usually melt snow within a few days to weeks. The climate is very dry, so the summer heat, typically 85 to the rare 100+ degree days, is very tolerable. Our one weather detractor is the wind. We have it frequently, and when it blows it is not unusual to experience gusts in excess of 45 mph. But this was a clear, quiet days, so it was unlikely we’d have chunks of windswept manure and sand landing on our sterile field.

We laid down some fresh shavings and put a clean sheet on the ground. Fern held the foal in a classic “foal grip” with one hand grapsing the base of the tail, her other arm looped under the foals neck, holding the head snuck against her chest. I sedated the foal using 50 milligrams of intravenous xylazine. Within three minutes she was a wet noodle, and we laid her down on the sheet, Tanya sitting at her head and my assistant Fern holding the affected leg in elevation. After wrapping the hoof in clean gauze and performing a sterile prep of the leg from well above the joint to hoof, I donned sterile gloves and quickly inserted the needles into the joint on either side. We were half-way through the flush procedure using a 1 litre bag of lactated ringers solution when a coyote stuck it’s head around the corner of the shed and stared at us.

“Chloe, you git!” snarled Tanya.

“My God, Tanya, is that a coyote?” I asked, trying not to lose my concentration.

“Heck no, that’s Chloe, she came from the pound in San Diego. My husband says he’s gonna divorce me if I don’t get rid of her, we’ve got so many dogs. You want her? “

“No thanks. Hey, let’s pay attention here, this foal is going to wake up soon.”

The foal responded well to a series of three joint flushes and systemic antibiotics. Her x-rays showed no evidence of invasion of the infection into the bone, and a sample of joint fluid obtained after the third lavage procedure indicated that the infection was resolving. What didn’t resolve was Chloe’s fascination with veterinary medicine, and on our last visit to see the foal the little coyote dog hopped into my truck and made it very clear that she belonged there for better or worse. I changed her name to Sticky after a few weeks because she stuck to me like a little flea, and over the years she became as much a part of my practice as my stethoscope.

Eight years later, as I’m speeding along, speaking with the UC Davis resident about Brandy and rubbing Sticky’s ears, my fingers discover a lump under Sticky’s jaw. When we arrive at our next call I take a closer look at my little dog with the piercing topaz eyes. Not only can I palpate an enlarged lymph node at the base of her neck, when I examine her oral cavity I discover a dense mass of tissue covering the floor of her mouth between her lower canines.

“Oh shit, Fern, look at this - I think Sticky has cancer,” I say quietly.

Bad news.

Wednesday, February 3, 2010

Wednesday, Feb 3, 2010 - the Carpus (Knee)

http://www.highdesertequine.com/
Vet Tip of the Day:
Key words - horse knee conformation, carpus anatomy

Today we'll take a quick look at anatomy then focus on conformation. The equine knee, or carpus, is anatomically related to your wrist. It is located in the center of this radiograph, and as you can see, it contains two rows of bones. Therefore there are three joints in the carpus, from top to bottom they are the radiocarpal joint, the intercarpal joint, and the carpometacarpal joint. Review: Remember when discussing the cannon bone and splint bones they were related to the metacarpal bones of your hand (between your wrist and knuckles).  In the lower half of this radiograph you can see the cannon bone (metacarpal III) centrally, with the medial and lateral splint bones (metacarpals II and IV) on either side. While a common site of fracture in race horses, lameness directly related to the knee is less common in lower impact performance horses. However, when evaluating a horse for purchase or suitability for athletic performance, it is important to be able to assess a horse's knee conformation accurately.

There are several common  faults that occur at the knee. Looking at the diagram on the left, you will notice the leg on the left is "back at the knee" or "calf-kneed", the middle leg is correct, and the leg on the right is "over at the knee" or "buck-kneed". Obviously, the horse with the straight conformation (in the middle) is most desirable.

Another common conformational fault in knees is the "Bench knee" or "Offset knee".  Looking at the horse from the front, if you draw a straight line down the center of the forelimb, continuing through the middle of the knee and down to the ground, in a bench-kneed horse that line will not continue through the center of the cannon bone, pastern and hoof below the knee. This horse's leg below the knee is offset to the outside, so that the vertical line passes along the inside edge of the cannon bone, instead of the middle. This results in an imbalanced leg with more weight bearing through the inside splint bone which predisposes the horse to develop "splints" or excessive bone production along the inside or medial splint bone.

Tommorrow is Thursday, so look forward to this week's High Desert Veterinary Tale: Good News/Bad News.

Tuesday, February 2, 2010

Tuesday, February 7: The Fetlock and Metacarpus



Vet tip of the Day:

First a quick review: we've looked at the anatomy of the bones in the pastern and foot and the attachment of the deep flexor tendon onto the back of the second pastern bone and coffin bone. We've discussed how irregularities in the navicular bone can contribute to heel pain by irritating the flexor tendon and navicular bursa as the tendon slides up and down against the back of the navicular bone. And we've reviewed ring bone, an arthritic condition that occurs at the P1-P2 and P2-P3 joints.

Today we will move on to the bones and major soft tissue structures between the knee (carpus) and the pastern. The cannon bone, with a much smaller splint bone on either side, spans this distance. The bottom of the cannon bone rests on the top of P1, the long pastern bone, and forms the fetlock joint. The cannon bone and the splint bones are all metacarpal bones, which correspond to the bones in your hand between your wrist and your knuckles. At the level of the fetlock joint, just behind the cannon bone, lie the sesamoid bones. The suspensory ligament, which arises from the back of the knee, lies along the back of the cannon bone and splits just above the sesamoids, with one branch attaching to the top of each sesamoid bone. The deep and superficial flexor tendons originate behind the knee and attach at the level of the pastern. The deep flexor tendon attaches to the back of P1 and P2 (the long and short pastern bones), and the superficial tendon to P2 and P3 (the short pastern and coffin bones).

The distal, or lower cannon bone, suspensory branches and fetlock joint are like an amazing suspension bridge which is critical in supporting the weight of the horse during exercise. They are very common sites of injury in athletic horses. You may have seen pictures of the x-rays of Barbaro, whose catastrophic injury involved not only a fracture of the end of the cannon bone, but also his sesamoid bones, suspensory ligament and pastern. In our less extreme athletes, small strains in the suspensory branches, chip fractures in the fetlock, and inflammation of the sesamoid bones are all relatively common causes of lameness.

Pick up your horse's right front leg and hold the foot in your left hand. Place your right thumb on the inside, and your index finger on the outside, of the cannon bone mid-way between the knee and the fetlock. Squeeze firmly and let your fingers slide over the structures moving toward the back of the leg. The suspensory ligament is difficult to feel because it is very firm and lies closely attached to the back of the cannon bone. However, it is easy to feel the deep and superficial digital extensor tendons which lie just under the skin. Learn to distinguish between them, and then move your hand up and down the leg, feeling the length of each tendon from knee to fetlock. Just above the fetlock, on the inside and outside, it is possible to clearly palpate the branches of the suspensory ligament where they attach to the top of the sesamoid bones. Your hands are very sensitive indicators of changes in the soft tissue structures of the leg that can alert you to the presence of injuries.

Ask a Horse Vet Online

We have partnered with JustAnswer so that you can get an answer ASAP.

JustAnswer